insomnia-clients.org

Diagnosis - treatment


Sorry to say so, but physicians are very political animals. Often they confuse 'diagnosis' with 'solution' - like politicians in election campaigns. We prefer the view of engineers and exact scientists.

Most readers will say 'yes, I once had a bad night for no reason, that goes over'. That is correct - in that case. BUT: is may also go wrong.

Usually is goes wrong for a human and short term reason, as stress, being in love, etc - see our checklist:

Until now physicians had a checklist like: sleeping problems:
1. temporary? Prescribe low dosis for few weeks, it goes over.
2. Longer than a week? Analysis of factors from outside, in life,
a): worries? financial worries? fears? lonely? trauma's? in love?
b): physical discomfort: wrong bed? disturbed by smell, noise, temperature, touch, food, drink?
c) comorbide with other illnesses?

In case it was 'not all that', a physician once invested 'NLS', Nervous Legs Syndrome, something no patients complaints about, but physicians did not want to say 'don't know'.

Since the 20th century, far more is known about the inner brain working of sleep, and the following reasons for insomnia exist as well:

5 neurological reasons for chronic insomnia

1) lesions, especially in the VLPO
2) no neuron firing by the VLPO to generate Slow Wave Sleep (SWS)
3) not enough ion channels go open in the VLPO
4) to many other transmitters are being bounded by VLPO neurons, instead of GABA
5) not enough endogenous benzodiazepines (! the own body also makes benzodiazepine, so it is as natural as you wish).

The next article about CatSleep show why many things can already go wrong in the brain. 'Lesions', so fractures in tissues, are the number 1 reasons ofr people having chronic insomnia. Lesions can develop 'on itself' or be triggered by trauma. See 'Von Economo' for lesion analysis, and this lesion article: Saper lesions VLPO 2000 (PDF), Also in his famous 2005 description Dr. C. Saper refers to the 'fatal insomnia', that was already found in the 18th century.....but physicians prefer to hide these kinds of secret as you read:

Above neurological analysis to CONFIRM long term insomnia reasons, has not been worked out well yet. Although it would largely be possible with:

  • MEG scanners (Magento Ence Phalography)
  • fMRI
  • chemical technques, especially for 4) and 5).

    It seems that neurologists are very much interested in VISIBLE illnesses, and not in illnesses that cannot be seen (but only be measured, as nigh no sleep = night no SWS = Slow Wave Sleep on the EEG or the MEG).

    The chronic insomnia complaints of patients, with no doctors giving effort to investigate wether all 'Saper flip flop circuit elements are in order' in order to sleep well, leads to longterm repeated conflicts between long term insomniacs and physicians.

    Long term insomniacs, with insomnia as primary disease, very probably have an inner-brain disturbance (as the 5 neurological reasons mentioned) - but a doctor, who follows clinical guidelines about as much as a call centre script (without knowing the background in this case) stick to a few weeks of moderate sleeping pills, or eventually: moderate long term - but quite often a too low portion to perform. With 4-5 hrs of sleep AND complaints one is likely to perform less, being dismissed for that reason, miss a partner for that reasons, skips children (for management/stress reasons) - a shortage of sleeping pills can be very cruel and can wreck a life.

For people with inner brain chronic insomnia, the sleeping drug is 'therapeutic' in the sense that it heals; it is the dose required to get through the day, making a living and SCORING due the day, because that is expected from you. Nobody excuses you 'O, you have a lesion, you may be a bit more irritated'.

So chronic insomniacs will rather have a heavy dosis, without this increasing or making ''dependent' at all. It is just a stable long term dos.

For anxiety, alprazolam or xanax is set at: up to 4 mg per day (see also http://www.pdrhealth.com/drugs/xanax) http://www.psychtreatment.com/xanax_dosage.htm


Conclusion for now:

 

It leaves us with the conclusion, for chromic insomniacs:
a) either keep on struggling for decades with physicians in order to get more sleeping pills because they did not know Saper's fliflop, or did not study it, or did not measure it in your case
b) beg the neurologists to check on the 5 potential neurological disorders in the flipflop model:

1) lesions, especially in the VLPO
2) no neuron firing by the VLPO to generate Slow Wave Sleep (SWS)
3) not enough ion channels go open in the VLPO
4) to many other transmitters are being bounded by VLPO neurons, instead of GABA
5) not enough endogenous benzodiazepines (! the own body also makes benzodiazepine, so it is as natural as you wish).

Chances are however that the neurologists are not motivated, because a) it is not visible (neurologists LOVE to have visible effects) b) it is not the 'neurologists illness' by tradition...it would take them decades to struggle or get used that 'insomnia', at least 'chronic insomnia' is neurologists territorium insteand psychiatrists.

c): JUMP to something that works! Go to http://sleepgenerator.info to wee how we can arrange for you:
1) a wireless neurostimulator that stimulates your VLPO and therefore induces sleep
2) an EM wave masq that introduces synchronized EM waves that facilitate the low wave sleep.






Feel free to surf around, feel fee to email us on info@insomnia-clients.org and, as we have to pay all research our selves, we are totally dependent on private donations

account: 7599262 (ING bank, Netherlands)
beneficiary: Insomnia Clients Foundation
Amsterdam. Netherlands
IBAN: NL07INGB0007599262
BIC: INGBNL2A



Falling asleep is a mechanism in the brains....from central to decentral nervous system