insomnia-clients.org

Insomnia - neurotransmitters - emotions

 

As you probably now, neuroscience is revolutionising psychology and neurology since appr. 2005. For your own research, we like to give you a headstart bij giving you essential tables. From here, with help of google.scholar.com, you should be be able to conduct insomnia research in several directions.

The 'name of the game' in the brains is only half about 'physiology', a cross-field between biology and chemistry. It is at least 50% electronics: neurons firing pulses to the next, and neurons being very sensitive to electro magnetic winds, especially varying electro magnetic fields.

We will show you two tables:

1) per (essential) neurotransmitter, what the involved EMOTIONS and MOTORY functions are
2) per emotion, the relevant AREA IN THE BRAIN
This way it is easy to make connections in your research.

For example: isomnia has mostly to do with the neurotransmitter GABA. GABA is also the main neurotransmitter responsible for FEAR. Increased anxiety (and inhibited anxiety suppression) take place in the OFC and PFC.

With above three lines, your research can be quite effective and focussed.
Some things are of course 'non-emotional', so the generation of sleep out of the VLPO (a) producing and distributing GABA b) generation of the neural synchronisation in SWS, Slow Wave Sleep) has little to do with emotion. But the onset of sleep can be hindered by the emotion FEAR.
(a summary table is HERE - the total table 1 in PDF is here)

Table 2 gives the emotion deficit and then area of damage in the brain. Finally, some citations are given.

It is clear that the OFC, Orbito Frontal Cortex, is key to almost all emotion regulation deficits (!). One would wish a 'reset of OFC' (or 'replacement') would be possible.

Conclusion

With regard to insomnia, emotion number 9, FEAR, is most important. For fear as well as insomnia, the GABA neurotransmitter is essential. Much anxiety = decreased GABA level = insomnia. Regulation of fear happens in the OFC, secretion of GABA starts at the VLPO. Sleeping pills stimulate the binding of GABA to receptors; intentioned EM-waving of the OFC is the 'electric method' to decrease anxiety (as emotion) and increase sleep.

Four final remarks on fear.
1) fear is originally mentioned as 'shortcut' to save one's life. So it reacts FASTER than the whole rational way through 'thinking'. This is the more beneficial for less intelligent humans (they can survive because of fear instinct, despite slow thinking)
2) fear is not rational. Fear is associative reaction, not a rational one. Fear can neither be inhibited by rational thinking afterwards (!)
3) fear is a 'taboo' emotion: it is denied, hided, not seen. Look at scientific research on insomnia: who has made a link between insomnia and fear? Almost nobody between psychiatrist's her Freud and appr. 2005, the breakthrough of neuroscience.
4) according to the writings of ex-prisonned writers, philosopher's and 'heroes' having survived years of 'terror', fear is the only USELESS emotion, and the only inhibiting emotion. Fear in free nature is a totally functional and useful emotion. Fear in prison, continous war etc. is totally useless and a nightmare. Fear and insomnia will probably grow the coming decennia, as politics and voters make the choice: let systems crash and let people be flexible to make up for it individually. But human is not made to be the individual cushion of crashing systems (as banks, 2008).




Falling asleep is a mechanism in the brains....from central to decentral nervous system